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MIT Autism research - Reversal of symptoms?
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<blockquote data-quote="--Eleanor--" data-source="post: 55493" data-attributes="member: 3620"><p>The MIT study concerned only Fragile X syndrome, which is actually a very tiny subset of autism spectrum disorders, and one of the few subsets for which the "cause" actually is known. Fragile X generally affects boys, whose X chromosome (comes from the mother) has a specific abnormality. Since girls have two Xs, they generally are okay because one of them is "normal," but they can pass on the abnormal one to a son. Fragile X generally has both mental retardation and autistic traits. Since Fragile X can be very easily detected (and ruled out as a cause for most kids' autism spectrum disorders) this breakthrough isn't really making a lot of waves in the autism community, as far as I can tell.</p><p></p><p>As to the "cure" versus "don't cure" debate that rages on the internet, with the "neurodiversity" crowd jeering at the "curebie" crowd and vice versa, I generally stay out of it because it is premature. Nobody has come even close to actually finding a cure for autism (which, as noted above is a neurological disorder and not a disease). I think that what a lot of the anti-cure folks are concerned about is not that there will ultimately be a magic pill to cure autism, but that specific genetic markers will be identified so that autistics (or potential autistics) will be aborted--that is, autism will no longer exist as a natural part of neurodiversity. (Similar to what is happening with Down Syndrome.)</p></blockquote><p></p>
[QUOTE="--Eleanor--, post: 55493, member: 3620"] The MIT study concerned only Fragile X syndrome, which is actually a very tiny subset of autism spectrum disorders, and one of the few subsets for which the "cause" actually is known. Fragile X generally affects boys, whose X chromosome (comes from the mother) has a specific abnormality. Since girls have two Xs, they generally are okay because one of them is "normal," but they can pass on the abnormal one to a son. Fragile X generally has both mental retardation and autistic traits. Since Fragile X can be very easily detected (and ruled out as a cause for most kids' autism spectrum disorders) this breakthrough isn't really making a lot of waves in the autism community, as far as I can tell. As to the "cure" versus "don't cure" debate that rages on the internet, with the "neurodiversity" crowd jeering at the "curebie" crowd and vice versa, I generally stay out of it because it is premature. Nobody has come even close to actually finding a cure for autism (which, as noted above is a neurological disorder and not a disease). I think that what a lot of the anti-cure folks are concerned about is not that there will ultimately be a magic pill to cure autism, but that specific genetic markers will be identified so that autistics (or potential autistics) will be aborted--that is, autism will no longer exist as a natural part of neurodiversity. (Similar to what is happening with Down Syndrome.) [/QUOTE]
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